Eleven out of a series of twenty-nine patients (37-9%) with acute copper sulphate poisoning developed acute renal failure. Should copper sulfate be ingested, it’s only mildly toxic as it’s most often vomited up relatively quickly due to the extreme irritation it causes on the gastrointestinal tract. Copper sulfate is a corrosive acid used as a pesticide in agriculture, a preservative in the lumber industry, and as an algaecide. 5 The toxicity of copper sulfate depends on the copper content. Steve Savage, a San Diego, California-based geneticist, remarked that “copper sulfate was a state-of-the-art fungicide, in 1885!” Special mention should be made of copper sulphate poisoning.

Copper sulfate has a history. Hemolysis and hemoglobinuria may develop after 3 days if the animal survives the GI disturbances. Intravascular haemolysis appeared to be the chief factor responsible for renal lesions in these patients. Copper is an essential mineral. Copper poisoning through the skin is rare, as compresses with a solution of copper salts are not widely used. Topical application of a 1% soln is of value for phosphorus burns of the skin. It can, however, kill a whole tree if it is properly applied. 4,5 It is available as a bright blue crystal that is commonly referred to as blue stone or blue vitriol. Histological lesions observed in the kidney varied from those of mild shock to well established acute tubular necrosis. Copper sulfate poisoning is rare in the Western world. It can kill bacteria, algae, roots, plants, snails, and fungi.
Copper sulphate ingestion (accidental or deliberate) is a rare form of poisoning usually limited to the Indian subcontinent. Dry matter does not have a harmful effect, and accidentally ingested solution can be quickly removed with ordinary water. Ingestion of 15–20 mg of copper sulphate is reported to cause mainly gastrointestinal symptoms, with higher doses resulting in multi-organ damage. Lethal doses of copper sulphate ingestion are 10–20 g.1, 2. Though the rates are on the decline, it is essential that physicians are aware of its lethal complications and management strategies. Copper sulphate is a poison often used to discourage tree roots from growing into vulnerable areas such as sewer lines. However this may not be apparent always, as if the patient is unconscious or unable to give a history. Patients can present with a known history of copper sulphate poisoning, if they complaint or if there is an eye witness. The sudden onset of clinical signs in chronic copper poisoning is associated with the hemolytic crisis. Copper sulfate poisoning in high-dose ingestions and ). Clinical manifestations Common clinical manifestations of copper sulfate poisoning include erosive gastropathy, intravascular hemolysis, hepatitis, acute kidney injury and hemoglobinuria (2-8). It can be found in the environment, foods, and water. In this paper, we presented a patient who died despite early supportive and specific treatments. If someone consumes copper sulfate and does not vomit, they could be at risk of copper sulfate poisoning.

Gastrointestinal symptoms predominate in copper sulphate toxicity include: vomiting, haematemesis, melaena, and diarrhoea. What is copper sulfate? Copper sulphate poisoning .


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